PCOS Fertility

PCOS is one of the many reasons of female infertility, and can be resolved if managed mindfully. Unived’s PCOS Fertility is formulated with a clinically studied and proven 3.6:1 ratio of Myo-inositol to Caronositol® (Natural D-Chiro-Inositol), which has shown a 65.52% fertility success rate in clinical trials.

KEY BENEFITS

  • Supports female fertility and reproductive health.
  • Supports healthy ovarian function.
  • Helps increases chances of fertility by restoring inositol balance
  • Higher DCI concentration to compensate for epimerase inactivity
  • Helps reduces hyperinsulinemia, hyperandrogenism, LH levels to help restore ovulation
  • Helps regularize healthy menstrual cycle
  • Helps reduce symptoms of PCOS.

 1,850.00 4,995.00

  • 1 Month Supply
  • 3 Month Course
Clear
i
Batch No: D.F.103.1 MFG: 13.Oct.20 EXP: 13. OCT.22
Check COD Availability


Check

Ingredients Per Serving

Myo-Inositol – 1100mg, Caronositol® (Natural D-Chiro-Inositol 97% from Carob pods) – 300mg, Folate (as L-5 Methyltetrahydrofolate) – 400mcg

Delivers a 3.6:1 ratio of Myo-Inositol (1100mg) : D-Chiro-Inositol (Caronositol® – 300mg).

Serving Size

Two Capsules

Nutrition Facts

UNIVED PCOS FERTILITY NUTRITIONAL FACTS

Benefits

Unived’s PCOS Fertility contains Myo-Inositol and Caronositol® which is natural Plant- based D-chiro-inositol in a ratio 3.6:1. This ratio provides a higher concentration of DCI along with MI, and has been proven to support fertility in PCOS women.

We’ve also included 400mcg folate in its most active form L-5 Methyltetrahydrofolate to support pregnancy and lower homocysteine levels.

DCI and MI together are known to lower Insulin resistances, Insulin levels, Hyperandrogenism, LH and LH/FSH ratio levels all this together work to promote ovulation. Ovulation in presence of higher DCI to maintain the MI/DCI balance naturally creates a more sustainable environment to hold pregnancy.

Key Benefits

  • Helps improve menstrual disorder and regularize menstrual cycles
  • Helps reverse anovulation
  • Helps increases chances of fertility by restoring inositol balance
  • Higher DCI concentration to compensate for epimerase inactivity
  • Helps reduce insulin levels and increase insulin sensitivity
  • Helps reduce total and free testosterone levels
  • Helps reduce elevated luteinizing hormone levels
  • Helps maintain healthy luteinizing hormone/Follicle stimulating hormone ratio
  • Helps reduces symptoms like Hirsutism, Acne, alopecia and insulin resistance

Product Description

Description

3.6:1 or 40:1 – Ratio or Concentration?

Administration of exogenous DCI may be a means of bypassing defective epimerase activity and achieving the downstream metabolic effects of insulin in DCI‑deficient tissues. Epimerase activity is unidirectional, and DCI administration alone will not be able to mimic the effects of MI. Therefore, it is rational to provide a combination of both to ensure optimal insulin sensitivity[v].

While MI is necessary for metabolic management, DCI is equally important for menstrual, ovulatory, and cutaneous hyperandrogenic resolution. Therefore, the ratio may be less important than the absolute concentrations of both inositols. It is clear, therefore, that a high concentration of DCI is necessary to circumvent epimerase deficiency and ensure adequate levels in the ovary[v].

  • A study was performed to evaluate the efficacy of MI and DCI ratios for increasing the fertility rate. The primary outcome was the pregnancy rate, and the secondary outcomes were oocyte maturation, embryo quality, testosterone levels and insulin sensitivity[vi].
  • This was a multicenter controlled, randomized, double-blind parallel group study with two MYO-DCI formulations for 12 weeks. The study group (SG) was administered 550mg of MYO + 150mg of DCI twice daily; the control group (CG) was administered 550mg of MYO + 13.8mg of DCI twice daily[vi].
  • The participants comprised 60 women with PCOS undergoing ICSI. At baseline, no differences were found between the two groups regarding age, BMI, HOMA-IR or testosterone levels6.
  • The pregnancy and live birth rates were significantly higher in the SG than in the CG (65.5 vs. 25.9 and 55.2 vs. 14.8, respectively)[vi]
  • The combination of MYO-DCI at high doses of DCI improves the pregnancy rates and reduces the risk of OHSS in women with PCOS undergoing ICSI[vi].
  • The primary findings of this study show that high doses of DCI combined with MYO increase the percentage of pregnancy rates in women with PCOS undergoing ICSI[vi].

MYO and DCI can improve the outcomes by diverse mechanisms: improving insulin sensibility, increasing ovulation or reducing oxidative stress of follicular fluid6. The main strength of this study is that the pregnancy rates are high with 150 mg of DCI twice daily and could involve DCI in early embryonic implantation and development6. Thus we can say that higher DCI helps maintain an environment to support implantation of embryo and increases the chances of pregnancy and live births.

3.6 TO 1 RATIO FERTILITY STUDYWhy Unived’s PCOS Fertility?

Unived’s PCOS Fertility product is formulated with Myo-Inositol and Caronositol® D-Chiro-Inositol and folate in it bioactive form. Caronositol® is a natural Plant-Based DCI from carob extract obtained by a patented process in a ratio of 3.6:1 MI:DCI which is way higher in concentration of DCI as compared to the physiological ratio of 40:1. DCI helps reduce hyperandrogenism and skin disorders in women suffering with PCOS. It helps PCOS affected women by regulating normal ovulation.

We created PCOS Fertility using the two inositols in this ratio because we had to believe in facts and that is; in PCOS,

  1. MI to DCI conversion is impaired
  2. DCI is deficient
  3. DCI deficiency increases insulin resistance
  4. This leads to DCI paradox in the ovaries i.e. more MI to DCI conversion and excessive release of androgens.
  5. 3.6:1 ratio has shown an increase in pregnancy rates and live birth rates.

INOSITOL DIAGRAMThus is just makes more sense to supplement with a higher amount of DCI in combination with MI if one with PCOS is aiming to conceive.

Although Unived’s PCOS Fertility does not guarantee pregnancy to you because pregnancy could be affected by a number of factors depending on both partners, but it will help reduce the factors related to PCOS that might be hindering conception.

Additional information

Pack of

1 Month Supply, 3 Month Course

What is PCOS

Polycystic ovary syndrome (PCOS) is the most common endocrine disorder in women and a major cause of anovulatory infertility[1] Polycystic ovary syndrome (PCOS) is a complex heterogeneous disorder that has several aspects in terms of pathology such as metabolic, endocrine, reproductive, and psychological[2]. It is the most common endocrine diseases that affects 5 to 10% of women of adolescent and reproductive age[2].

There is increasing evidence to suggest that PCOS affects the whole life of a woman. It can begin in utero in genetically predisposed subjects, it manifests clinically at puberty, and continues during the reproductive years[1]

Several studies suggest that insulin resistance and hyperandrogenism play a central role in the progression of PCOS pathophysiology. Therefore, common treatment strategies of PCOS are based on lifestyle modification, which include exercise, diet, and nutrient supplementation therapy[2]

Normal Menstrual Cycle VS PCOS Cycle

The menstrual cycle is regulated by hormones. Luteinizing hormone (LH) and follicle-stimulating hormone (FSH), which are produced by the pituitary gland, promote ovulation and stimulate the ovaries to produce estrogen and progesterone. Estrogen and progesterone stimulate the uterus and breasts to prepare for possible fertilization. The menstrual cycle has three phases:

  • Follicular (before release of the egg)
  • Ovulatory (egg release)
  • Luteal (after egg release)

In a Healthy Menstrual cycle, when the follicular phase begins, levels of estrogen and progesterone are low. As a result, the top layers of the thickened lining of the uterus (endometrium) break down and are shed, and menstrual bleeding occurs. About this time, the follicle-stimulating hormone level increases slightly, stimulating the development of several follicles in the ovaries. Each follicle contains an egg. Later in this phase, as the follicle-stimulating hormone level decreases, only one follicle continues to develop. This follicle produces estrogen.

The ovulatory phase begins with a surge in luteinizing hormone and follicle-stimulating hormone levels. Luteinizing hormone stimulates egg release (ovulation), which usually occurs 16 to 32 hours after the surge begins. The estrogen level decreases during the surge, and the progesterone level starts to increase.

During the luteal phase, luteinizing hormone and follicle-stimulating hormone levels decrease. The ruptured follicle closes after releasing the egg and forms a corpus luteum, which produces progesterone. During most of this phase, the estrogen level is high. Progesterone and estrogen cause the lining of the uterus to thicken more, to prepare for possible fertilization.

If the egg is not fertilized, the corpus luteum degenerates and no longer produces progesterone, the estrogen level decreases, the top layers of the lining break down and are shed, and menstrual bleeding occurs (the start of a new menstrual cycle).REGULAR MENSTRUAL CYCLE -FERTILITY

MENSTRUAL CYCLE WITH PCOS

In PCOS, the cycle is hindered right in the follicular phase. The follicle stimulating hormone increases for follicle maturation but;

  1. With PCOS, LH levels are often high when the menstrual cycle starts. The levels of LH are also higher than FSH levels.
  2. Because the LH levels are already quite high, there is no LH surge. Without this LH surge, ovulation does not occur, and periods are irregular.
  3. Girls with PCOS may ovulate occasionally or not at all, so periods may be too close together, or more commonly too far apart. Some girls may not get a period at all.

A high percentage (55–75%) of women with PCOS have an elevated LH/FSH ratio presumably due to high levels of LH rather than reduced production of FSH. Higher LH further leads to excessive androgen production and the ovaries may develop numerous small collections of fluid (follicles) and fail to regularly release eggs. Inositol imbalance consisting of excess myo-inositol and deficient chiro-inositol is a measure of insulin resistance this is because of epimerase inactivity in PCOS. All this could be a result of many factors.

PCOS Symptoms

Main Symptoms

  1. Menstrual disorder – oligomenorrhea or amenorrhea meaning irregular periods or no periods at all.
  2. Reproductive concerns/Anovulatory cycles – The majority of women with PCOS have anovulation. With this comes infertility as well as problems of dysfunctional bleeding[3]. Perhaps the most frustrating reproductive concern for women with PCOS is pregnancy loss. The spontaneous abortion rate in PCOS is approximately one third of all pregnancies3.
  3. Hirsutism – presence of terminal hair on the face and/or body in a masculine pattern. It is the most common symptom, found in about 60 % of women with PCOS[1].
  4. On diagnosis – presence of 12 or more follicles of diameter 2–9 mm or an ovarian volume of more than 10 mL in follicular phase[1].
  5. Obesity – not all women with PCOS are obese and not every obese woman has PCOS. But when present, obesity worsens the clinical presentation of PCOS increasing insulin resistance and resulting in a further elevation of ovarian and adrenal androgens and of unbound testosterone3.
  6. Impaired glucose tolerance & diabetes – All women with PCOS are therefore at risk to develop impaired glucose tolerance and overt type 2 diabetes3. In a recent study, impaired glucose tolerance was found in 31% of women of reproductive age, with PCOS and diabetes in 7.5%3.
  7. Acne
  8. Alopecia – consists of progressive hair loss or thinning Hair loss in PCOS usually involves thinning at the vertex with maintenance of the frontal hairline[1].

Factors responsible for PCOS

  1. Altered secretion of GnRH and gonadotropins: GnRH produced by the Hypothalamus enters the anterior pituitary gland and cause it to produce two important hormone; FSH and LH. GnRH over stimulation causes, indeed, excessive LH production[1].
  2. Ovarian and extraovarian hyperandrogenism: Hyperandrogenemia is the most typical hormonal alteration of PCOS. Hyperandrogenism has a multifactorial origin attributed mostly to the ovaries with a substantial contribution from the adrenals and a minor contribution from fatty tissue. Aromatase is the enzyme that converts androgens into estrogens. It may be responsible for hyperandrogenism as Low aromatase activity has also been demonstrated in women with PCOS[1].
  3. Hyperinsulinemia and insulin resistance: Since many women with PCOS seem to have insulin resistance, compensatory hyperinsulinemia is thought to contribute to hyperandrogenism by direct stimulation of ovarian production of androgens and by inhibition of liver synthesis of SHGB that increases testosterone availability. Insulin also increases ACTH mediated adrenal androgen production[1].
    1. The action of insulin on the ovary uses the inositol glycan system as a signal mediator, a different mechanism from the system activated by phosphorylation of the receptor at tyrosine level in other tissues. An increase was observed in urinary clearance of inositol in some American and Greek women with PCOS. It reduces tissue availability of inositol. This mechanism could contribute to insulin resistance present in PCOS women[1].Briefly, insulin is considered as a key hormone for hyperandrogenism in the PCOS pathophysiology via two different pathways:
      • Insulin stimulates androgen production of theca cells with luteinizing hormone (LH) and elevated androgen production leads to hirsutism, acne, and anovulatory infertility.
      • Hyperandrogenism associated function of insulin is inhibition of sex hormone-binding globulin (SHBG) synthesis in the liver.
  4. Anti-Mullerian hormone (AMH): Women with PCOS have higher serum and Follicular Fluid concentrations of AMH compared to controls. This is closely correlated with greater development of antral follicles and arrest of follicular growth. High serum levels of AMH are directly correlated with an increase in testosterone and/or LH concentrations in women with PCOS, as well as with altered oocyte maturation and low embryo quality[1].
  5. Other factors like unhealthy diet or eating habits, inactive lifestyle, environmental factors or genetics can also contribute in the development of PCOS.

Although the etiopathogenesis of PCOS is still controversial, mentioned above are some hypotheses that have been proposed in the recent decades[1].

Stages

According to the Rotterdam criteria, PCOS is confirmed by the existence of at least two out of three criteria

  1. Hyperandrogenism,
  2. Chronic anovulation,
  3. Polycystic ovaries on ultrasound findings

Clinical Manifestation

The typical clinical indications of PCOS are: anovulatory cycles, ultrasonographic evidence of polycystic ovaries and hirsutism1. Many women are also overweight or obese and have an increased risk of developing metabolic syndromes in later life1. During pregnancy, there is a higher chance of miscarriage, gestational diabetes and hypertension1.

Factors that may be responsible for PCOS

  1. Altered secretion of GnRH and gonadotropins: GnRH produced by the Hypothalamus enters the anterior pituitary gland and cause it to produce two important hormone; FSH and LH. GnRH over stimulation causes, indeed, excessive LH production[1].
  2. Ovarian and extraovarian hyperandrogenism: Hyperandrogenemia is the most typical hormonal alteration of PCOS. Hyperandrogenism has a multifactorial origin attributed mostly to the ovaries with a substantial contribution from the adrenals and a minor contribution from fatty tissue. Aromatase is the enzyme that converts androgens into estrogens. It may be responsible for hyperandrogenism as Low aromatase activity has also been demonstrated in women with PCOS[1].
  3. Hyperinsulinemia and insulin resistance: Since many women with PCOS seem to have insulin resistance, compensatory hyperinsulinemia is thought to contribute to hyperandrogenism by direct stimulation of ovarian production of androgens and by inhibition of liver synthesis of SHGB that increases testosterone availability. Insulin also increases ACTH mediated adrenal androgen production[1].
    1. The action of insulin on the ovary uses the inositol glycan system as a signal mediator, a different mechanism from the system activated by phosphorylation of the receptor at tyrosine level in other tissues. An increase was observed in urinary clearance of inositol in some American and Greek women with PCOS. It reduces tissue availability of inositol. This mechanism could contribute to insulin resistance present in PCOS women[1].Briefly, insulin is considered as a key hormone for hyperandrogenism in the PCOS pathophysiology via two different pathways:
      • Insulin stimulates androgen production of theca cells with luteinizing hormone (LH) and elevated androgen production leads to hirsutism, acne, and anovulatory infertility.
      • Hyperandrogenism associated function of insulin is inhibition of sex hormone-binding globulin (SHBG) synthesis in the liver.
  4. Anti-Mullerian hormone (AMH): Women with PCOS have higher serum and Follicular Fluid concentrations of AMH compared to controls. This is closely correlated with greater development of antral follicles and arrest of follicular growth. High serum levels of AMH are directly correlated with an increase in testosterone and/or LH concentrations in women with PCOS, as well as with altered oocyte maturation and low embryo quality[1].
  5. Other factors like unhealthy diet or eating habits, inactive lifestyle, environmental factors or genetics can also contribute in the development of PCOS.

Although the etiopathogenesis of PCOS is still controversial, mentioned above are some hypotheses that have been proposed in the recent decades[1].

Stages

According to the Rotterdam criteria, PCOS is confirmed by the existence of at least two out of three criteria

  1. Hyperandrogenism,
  2. Chronic anovulation,
  3. Polycystic ovaries on ultrasound findings

Clinical Manifestation

The typical clinical indications of PCOS are: anovulatory cycles, ultrasonographic evidence of polycystic ovaries and hirsutism1. Many women are also overweight or obese and have an increased risk of developing metabolic syndromes in later life1. During pregnancy, there is a higher chance of miscarriage, gestational diabetes and hypertension1.

Who should consume PCOS Fertility?

PCOS Fertility is a product that will help manage the symptoms of PCOS by lowering the many features of the metabolic syndrome and help restore the physiological balance of MI and DCI which are impaired and resulting in problems. Once the right balance is achieved in the systemic circulation and the ovaries, the chances of fertility are higher.

This product is especially designed based on research to help the women for whom PCOS is the reason of infertility. However, conception is a very sensitive topic and can be affected by various factors.

If you are someone who is looking for something that helps regularize your cycles and manage symptoms associated with PCOS, but at this age & stage in life you may not be very focused on trying to conceive, then we recommend our PCOS Management product for you.

If you are someone who is looking for all of the above, but is also trying to conceive, then we recommend our PCOS Fertility product for you.

Please note, the PCOS Fertility product is formulated in a ratio that is clinically studied and has shown to deliver a higher chance of conception, while also addressing all the PCOS symptoms. Therefore, if you are at the age & stage in life where you not only want to address the symptoms but also want to try and conceive in the coming months, then we recommend choosing the PCOS Fertility product.

The PCOS Management product is formulated to address PCOS related imbalances, when these imbalances get addressed and your menstrual cycles resume, you do stand a fair chance to conceive. So, it is not that if you consume the PCOS Management product you will not have chances of conception.

It is just that one formulation offers a higher chance of conception as compared to the other.

If you are someone who has tried our PCOS Management and PCOS Fertility products, and found that PCOS Fertility works better for you in terms of regularizing your cycles, despite the fact that you are not planning for conception, you may continue using this product for regular PCOS Management as well.

We hope the suggestions are clear. Please feel free to speak with us if you require further clarification.

Mode of Action

Inositols

Two inositol isomers, Myo-Inositol (MI) and D-Chiro-Inositol (DCI) have been proven to be effective in PCOS treatment, by improving insulin resistance, serum androgen levels and many features of the metabolic syndrome[iv].

The enzyme epimerase converts MI to DCI, maintaining a physiological ratio of 40:1, which varies from tissue to tissue. The conversion rate of MI to DCI ranges from 7% to about 9% in normal healthy females and is much lesser in women with PCOS due to insulin resistance in the systemic circulation. In the setting of epimerase deficiency, less MI can be converted to DCI, a state of relative DCI deficiency occurs, and insulin resistance is promoted. This, in turn, leads to the metabolic complications of hyperinsulinemia[v].

Therefore to summarize; in the serum,

PCOS MODE OF ACTIONIn the ovary, MI and DCI have specific duties to perform. MI supports FSH signaling, whereas DCI is responsible for insulin-mediated testosterone synthesis. In the normal ovary, these activities proceed in balance, allowing the maintenance of normal hormonal levels and facilitating ovarian function. In the polycystic ovary, systemic insulin resistance (hyperinsulinemia) accentuates epimerase activity, thus creating a higher DCI-to-MI ratio. This promotes hyperandrogenism and reduces the efficiency of MI-mediated FSH signaling[v].

Diet Exercise & Lifestyle Modifications

PCOS FERTILITY LIFESTYLE CHANGESDiet and exercise are important parts of managing PCOS. Knowing the right foods to eat as well as the kinds of food to limit can improve the way you feel. Eating well, staying active, and maintaining a healthy weight (or losing even a small amount of weight if you’re overweight) can improve PCOS symptoms[vii].

PCOS FERTILITY DIET MODIFICATIONSExercise

It’s really important that girls with PCOS exercise, because exercise brings down insulin levels7,[viii] and can help with weight loss. Exercise can be especially helpful in lowering insulin after a meal8,7. So, if possible, go for a walk after you eat a large meal. Any increase in exercise helps, so find an activity, sport, or exercise that you enjoy. If you aren’t doing a lot of exercise now, start slowly, and build up to your fitness goal. If you only exercise once in a while, try to exercise more regularly. Work towards increasing your physical activity to at least 5 days a week for 60 minutes per day7,[viii].

A good fitness plan should include a balance of stretching, toning, and aerobic activities.

Weight Management Tips

  • Choose nutritious, high–fiber carbohydrates instead of sugary or refined carbohydrates
  • Balance carbohydrates with protein and healthy fats
  • Limit your portions when you’re eating high–carbohydrate foods (especially ones that are low in fiber), and try to eat them with foods that contain protein.
  • Eat small meals and healthy snacks throughout the day instead of large meals
  • Exercise regularly to help manage insulin levels and your weight

References

[1] V. De Leo, et. al., “Genetic, hormonal and metabolic aspects of PCOS: an update”, De Leo et al. Reproductive Biology and Endocrinology (2016) 14:38

[2] Elif Günalan, et. al., “The effect of nutrient supplementation in the management of polycystic ovary syndrome-associated metabolic dysfunctions: A critical review”, the Turkish-German Gynecological Education and Research Foundation Assoc 2018; 19: 220-32

[iii] Enrico Carmina And Rogerio A. Lobo,“Polycystic Ovary Syndrome (PCOS): Arguably the Most Common Endocrinopathy Is Associated with Significant Morbidity in Women”, The Journal of Clinical Endocrinology & Metabolism 1999 Vol. 84, No. 6

[iv] Antonio Cianci, et. al., “D-chiro-Inositol and alpha lipoic acid treatment of metabolic and menses disorders in women with PCOS”, Gynecol Endocrinol, Early Online: 1–4 2015 Informa UK Ltd.

[v] Kalra, Bharti et al., “The inositols and polycystic ovary syndrome”, Indian journal of endocrinology and metabolism vol. 20,5 (2016): 720-724.

[vi] Nicolas Mendoza, et. al., “Comparison of the effect of two combinations of myo-inositol and D-chiro-inositol in women with polycystic ovary syndrome undergoing ICSI: a randomized controlled trial”, Gynecological Endocrinology, 2019 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

[vii]  Phaedra Thomas RN, BSN, et. al., PCOS Resources for a Healthier You” ©Center for Young Women’s Health | Boston Children’s Hospital

[viii] Chris Kite, et. al., “Exercise, or exercise and diet for the management of polycystic ovary syndrome: a systematic review and meta-analysis”, Systematic Reviews (2019) 8:51

FAQ's

What is PCOS Management?

The best way to manage PCOS is to work  towards a healthy lifestyle that includes healthy eating and daily exercise and correct supplementation.

Does PCOS mean I have cysts on my ovaries? 

The term “polycystic ovaries” means that there are many tiny cysts, or bumps, inside of the ovaries. Some young women with PCOS have many cysts; others only have a few. Even if you do have many they’re not harmful and they don’t need to be removed14. 

Why are my periods so irregular? 

Having PCOS means that your ovaries aren’t getting the right (hormonal) signals from your pituitary gland. Without these signals, you will not ovulate (make eggs) every month. Your period may be irregular, or you may not have a period at all. 

Why do I get acne and/or extra hair on my body? What is hirsutism? 

Having hair on face and the body in a masculine pattern is termed as “Hirsutism” and very commonly observed in women with PCOS. Acne and extra hair on your face and body can occur if your body is overproducing testosterone. All women produce testosterone, but if you have PCOS, your ovaries produce a little bit more testosterone than they are supposed to. Skin cells and hair follicles can be extremely sensitive to the small increases in testosterone found in young women with PCOS14. 

What is insulin resistance? 

If your body is resistant to insulin, it means you need high levels of insulin to keep your blood sugar normal. Certain medical conditions such as being overweight or having PCOS can cause insulin resistance14. Insulin resistance tends to run in families. It also can increase blood glucose levels. 

What can insulin resistance do to me? 

High insulin levels can cause thickening and darkening of the skin (acanthosis nigricans) on the back of the neck, axilla (under the arms), and groin area. In young women with PCOS, high insulin levels can cause the ovaries to make more androgen hormones such as testosterone. This can cause increased body hair, acne, and irregular or few periods. Having insulin resistance can increase your risk of developing diabetes14. 

Are “carbs” (carbohydrates) unhealthy? 

No! Carbs (carbohydrates) are the basic source of energy. People often think that eating carbs will make them gain weight, but only too much of it will result in weight gain. Many other important nutrients come from carbohydrate foods, so eating no carbs is not a good idea. High–fiber carbohydrate foods are high in nutrients and help you feel full longer than sugary low-fiber carbohydrates, it’s best to choose these as often as possible. It is only a matter of quality and quantity. One should be mindful and make the right choice when selecting carbohydrates and prefer complex carbohydrates over simpler carbohydrates and should know how to portion it alongside fats and proteins. 

What about foods that have fats and proteins in them? 

High protein foods, and fats such as olive oil, nuts, and avocado are important parts of a PCOS–friendly diet. Combining foods that contain protein or fat with a carbohydrate will help to slow down the absorption of the carbohydrate and keep insulin levels low. For example, instead of plain rice, have rice with dal/beans and little veggies on the side. Keep in mind that some fats are much healthier than others.

How long will it take to see results? 

It is important to take PCOS Management daily for a period of 3-6 months to make the desired changes. Some individuals will experience results sooner than others as results depend on various factors, such as food and exercise as well.  

For how long is it safe to use Unived’s PCOS Fertility? 

PCOS is a syndrome that lacks cure and requires management throughout life. Unived’s PCOS Management can be consumed daily for a period of 6 months.

Should one expect pregnancy only because they started using Unived’s PCOS Fertility product?

Conception depends on a number of factors ranging from health status of both parents to the time of ovulation and many others. With our fertility product women with PCOS who want to conceive can be slightly more positive about ovulation occurring and reduced chances of miscarriage. Although we cannot guarantee a 100% chance of pregnancy we can still try to be the ray of hope for many women for whom PCOS is the cause of infertility.

Suggested Use

Adults take 2 capsules daily post lunch.

It is important to ensure you take a 3-6month course to see clear results. People who have been recently diagnosed and are not seeing severe symptoms may see results faster than someone who has a long history of dealing with PCOS.

  • Special Box
  • COA